Atlas of Ophthalmology

Male, 51 years of age, BCVA Light Perception, IOP 52 mmHg Neovascular glaucoma with an intraocular pressure of 52 mmHg after ischemic central retinal vein occlusion. The colour image showing an iris neovascularization. The resulting secondary glaucoma is due to a formation of a fibrovascular membrane on the anterior surface of the iris (rubeosis iridis). The membrane extends into the chamber angle and leads to irreversible obliteration of the outflow system, with a corresponding rise in intraocular pressure. The most common diseases responsible for development of NVG are ischemic central retinal vein occlusion (CRVO), diabetic retinopathy and ocular ischemic syndrome. Conventional treatments with extremely uncontrolled cases are retinal cryotherapy or enucleation. Regression of iris neovascularization by employing panretinal photocoagulation or intravitreal injection of anti-vascular endothelial growth factor antibodies create a chance to undertake antiglaucomatous surgery. Literature: (1) Hayreh SS. Neovascular glaucoma. Prog Retin Eye Res.2007 Sep;26(5):470-85. (2) Löffler KU. Neovascular glaucoma: aetiology, pathogenesis and treatment. Ophthalmologe. 2006 Dec;103(12):1057-63;

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